Manufacturing Snaps Back: Is it a Turning Point or ... - Yahoo! Finance

And just like that, a 3-month patch of manufacturing weakness has disappeared, carrying with it worries that we were headed towards another recession, while also giving stock market bulls a badly need shot of confidence on the heels of a 2 week retreat. The better than expected pop in September's ISM Manufacturing index is the type of single data point snapback that traders love, but it is also just that - a single data point - that professionals say will need to be corroborated in the weeks to come.

"It's a sigh of relief for the markets," says Jeff Kleintop, chief market strategist at LPL Financial. "Manufacturing is so important to the S&P 500, while it's only 20% of our economy, it's nearly 40% of S&P 500 profits."

In fact, data shows that over the past 50 years, there have been no less than 7 recessions in the U.S., and each them coincided with a plunge in the ISM Manufacturing to at least the mid-40s level. While that type of factory slowdown is to be expected, what isn't is the fact that there have also been a half dozen false positives or "flukes" Kleintop says, an issue that must be respected.

From his viewpoint, it is important to recognize that "manufacturing may not be as weak as we had thought" especially given the concurrent spike in new orders, which Kleintop calls the ''most important component."

"We don't usually get sharp reversals in this index. It usually trends lower for a year or so, then turns and rises," he says, adding that more data will be needed to confirm it.

The October ISM report comes out Thursday, November 1st -- the day before the final payroll and unemployment report is released and just 5 days before the election.

Source: http://finance.yahoo.com/blogs/breakout/manufacturing-snaps-back-turning-point-false-positive-164938094.html

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Microsoft set to get back in the news business with relaunched MSN

Microsoft set to get back in the news business with relaunched MSN

Microsoft isn't exactly a stranger to the news business. It founded Slate Magazine way back in 1996 as part of its then still-nascent MSN website, and it partnered with NBC that same year on the MSNBC cable news network. While it's since sold off both Slate and its stake in MSNBC, it looks like the company is now taking another big swing at producing its own news operation. As Reuters reports, Microsoft is making "big, multi-million dollar investment" to create what is being described as a "decent-sized media operation," one that will be a part of the new-look MSN that the company has also announced today. Details remain light beyond that, but Reuters says the new MSN will still "chiefly aggregate news" from traditional sources like Reuters and the AP in addition to producing its own content. As for that new MSN itself, it's unsurprisingly built with tablets (and Windows 8, specifically) in mind, with a design that's tailored for full-screen use and touch input. Its launch wil coincide with Windows 8's debut on October 26th, but you can get an early look at the link below and in the video after the break.

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tfs - Microsoft Visual Studio Team Foundation Server 2012 Power ...

I have upgraded our TFS 2010 (Win 2008 R2 SP1, SQL Server 2008 R2 SP1, WSS 3.0 SP2) to TFS 2012 (Win 2012 RTM, SQL Server 2012, SPF 2010 SP1). This is a single server farm (TFS, SP are installed on the same server). TFS build is installed on a separate server. Everything works fine.

In the TFS 2010 environment, I was able to use the TFS 2010 Power Tool to create a backup plan. I installed/upgraded the TFS power tool to TFS 2012 Power Tool.. When I try to create a backup plan using the backup plan wizard, I get errors. (my guess is during the upgrade some permission to the DB was revoked.)

This is my backup plan settings:

enter image description here

enter image description here

It looks like there are three issues:

1. Cannot find reporting databases

The following is logged:  [Info   @22:38:23.430] +-+-+-+-+-| Running VerifyReportingDatabases: Verify valid SQL Reporting Services database connection string |+-+-+-+-+-  [Info   @22:38:23.446]   [Info   @22:38:23.446] +-+-+-+-+-| Verify valid SQL Reporting Services database connection string |+-+-+-+-+-  [Info   @22:38:23.446] Starting Node: DataBaseAccess  [Info   @22:38:23.446] NodePath : Container/Progress/DataBaseAccess  [Info   @22:38:23.524] Node returned: Error  [Error  @22:38:23.524] Cannot find reporting databases  [Info   @22:38:23.524] Completed VerifyReportingDatabases: Error  [Info   @22:38:23.524] -----------------------------------------------------

2. Cannot find SharePoint databases for applications.

[Info @22:38:23.540] +-+-+-+-+-| Running VerifySharepointDatabases: Verify valid SharePoint database connection string |+-+-+-+-+-

[Info   @22:38:23.540]   [Info   @22:38:23.540] +-+-+-+-+-| Verify valid SharePoint database connection string |+-+-+-+-+-  [Info   @22:38:23.540] Starting Node: DataBaseAccess  [Info   @22:38:23.540] NodePath : Container/Progress/Conditional/DataBaseAccess  [Info   @22:38:26.965] Node returned: Error  [Error  @22:38:26.965] Cannot find SharePoint databases for applications:  http://xxxxxx-tfs01:17012/. This tool does not support multiple SharePoint farms in TFS configuration. If your TFS server is using multiple farms for SharePoint application, please exclude SharePoint component from the backup plan  [Info   @22:38:26.965] Completed VerifySharepointDatabases: Error  [Info   @22:38:26.965] -----------------------------------------------------   [Info   @22:38:26.965] +-+-+-+-+-| Running VerifyBackupEncryptionKeyOperation: Verify > Reporting Services encryption key can be backed up |+-+-+-+-+-  [Info   @22:38:26.965]   [Info   @22:38:26.965] +-+-+-+-+-| Verify Reporting Services encryption key can be backed up |+-+-+-+-+-  [Info   @22:38:26.965] Starting Node: VerifyBackupEncryptionKeyOperation  [Info   @22:38:26.965] NodePath : Container/Progress/Conditional/VerifyBackupEncryptionKeyOperation  [Info   @22:38:27.059] Node returned: Error [Error  @22:38:27.059] Provider load failure   [Info   @22:38:27.059] Completed VerifyBackupEncryptionKeyOperation: Error  [Info   @22:38:27.059] -----------------------------------------------------

3. Alert setup fails

[Info @22:43:57.295] +-+-+-+-+-| Running VerifyEmailNotifications: Verify Email Notifications |+-+-+-+-+-

[Info   @22:43:57.295]   [Info   @22:43:57.295] +-+-+-+-+-| Verify Email Notifications |+-+-+-+-+-  [Info   @22:43:57.295] Starting Node: VerifyEmailNotifications  [Info   @22:43:57.295] NodePath : Container/Conditional/Progress/VerifyEmailNotifications  [Info   @22:44:05.461] Node returned: Error [Error  @22:44:05.461] The Backup Task account 'xxxxxxx-TFS01\TFSServiceAccount' either does not have permissions on your SMTP host or the password specified is invalid. Select an account with permissions to send emails and confirm the password is correct.  [Info   @22:44:05.461] Completed VerifyEmailNotifications: Error  [Info   @22:44:05.461] -----------------------------------------------------

This was working in the TFS 2010 environment. enter image description here

Source: http://stackoverflow.com/questions/12682299/microsoft-visual-studio-team-foundation-server-2012-power-tools-db-backup-tool-e

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Non-Distress Home Sales Are Making A Come-Back in St Cloud ...

Non-Distress Home Sales Are Making A Come-Back in St. Cloud, Florida. You read that right. Non-Distress Sales. Not short-sales, not foreclosures, regular sales between a real seller and real buyer. The way Real Estate used to be. Buyers by the hand full who are fed-up and frustrated that banks juggle multiple offers on properties or wait for up-to a month to review an offer while the buyer lingers in limbo?and then seem to end up on the short end of the stick, are re-visiting traditional sales.

Short-Sales and Foreclosure Homes in St Cloud Florida,?are typically discounted seventeen to twenty-five percent (17-25%) off of fair market value. So a traditional sale may cost a little more. But, what a traditional sale offers that discount properties do not is a shorter response time to offers, and ability to negotiate.

In a Foreclosure sale, the bank knows NOTHING about the property they now own. Nothing. All the do know, is that borrower failed to make payments, and they went through the court system to take back possession of the property that the mortgage payments were defaulted on.

They have no idea how old the roof is, if it ever had a water leak, or when the carpet was installed. In addition, often those properties have been long neglected due to the harsh economic times and shortage of repairs funds most home owners set aside to keep up with routine maintenance.

What they do know, is how much they want to sell it for regardless of the amount of work it may need to repair it. They have done their homework. They have had it either appraised or obtained ?a Broker Price Opinion based on comparable sales in the area to determine a current market value.

So a bargain is not always a bargain.

?

?

?In addition to the defects you can see, there could be many you cannot see. They make no guarantee or warranty- it is sold "As-Is".?

?

? Having a home inspection performed may be one way to mitigate unexpected problems, but as any contractor knows one problem can often lead to another. It could become a money pit quickly.

In a traditional sale, the home owner has a better overview of repairs, problems, cures and dates work has been performed. There is something called a "Seller's Disclosure" which in Florida Home Sales, is a handy document covering a variety of topics all pertaining to the homes various systems. Ask for one.

? In the long run, a traditional sale, one that is not a distress sale may be worth a second look. The money you pay up front, may save you much more in repairs later. St Cloud Homes, LLC

St.Cloud Homes

Allison Stewart Broker, SFR, CDPE?

407-616-9904www.kissimmee-stcloudflhomes.com

???????????????????????????????????????????????????????????????????????????????????????????????????????

????St. Cloud Homes YOUR Local Real Estate Experts serving Home Buyers/Sellers in Kissimee, St. Cloud Fl, Harmony, Lake Nona, Anthem Park, Bay Lake Ranch, Lake Lizzie, Reunion, Celebration, Lakepointe, Steven Plantation, Acadia Estates, East Lake, Windermere, Bay Hill. Our unparalleled service begins with YOU.

?? For a free consultation call us 407-616-9904 or visit our website?or ?email us

?

Source: http://activerain.com/blogsview/3461610/non-distress-home-sales-are-making-a-come-back-in-st-cloud-florida

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Immune cells could protect against obesity, scientists discover

ScienceDaily (Oct. 1, 2012) ? New research has found that a type of anti-tumour immune cell protects against obesity and the metabolic syndrome that leads to diabetes. Results showing that immune cells known to be protective against malignancy called invariant natural killer T-cells (iNKT), that are lost when humans become obese, but can be restored through weight loss, have been published online this week in the journal Immunity.

Marie Curie Fellow, Lydia Lynch at Trinity College Dublin, Ireland made the discovery and as first author in collaboration with colleagues at Harvard-affiliated Beth Israel Deaconess Medical Centre, and St Vincent's University Hospital have shown that therapies that activate iNKT cells could help manage obesity, diabetes, and metabolic disease.

iNKT cells had been thought to be rare in humans until work by Dr Lydia Lynch at Trinity College Dublin, Consultant Endocrinologist at St Vincent's University Hospital Professor Donal O'Shea, and Trinity's Professor of Comparative Immunology, Cliona O'Farrelly, found they were plentiful in human omental fat.

"We then found a large population of iNKT cells in fat tissue from mice," said Dr Lynch whose Marie Curie Fellowship gave her the opportunity to work with Mark Exley and Steve Balk both assistant professors of medicine at Harvard Medical School (HMS) and leaders in the field of natural killer T-cell (NKT) investigations. "Now we have identified a role for these cells in the regulation of body weight and the metabolic state, likely by regulating inflammation in adipose tissue."

The team also discovered that a lipid called alpha-galactosylceramide (aGC) can lead to a dramatic improvement in metabolism, weight loss, and fatty liver disease, and can reverse diabetes by bolstering cells that have been depleted.

Dr Lynch first began this line of investigation in 2007 where her work with Professor Donal O'Shea in the Obesity Clinic at St Vincent's University Hospital in Dublin focused on the immune systems of obese patients. All human data was from Irish patients and carried forward to Boston. "We knew that not only did obese patients have more heart attacks and a greater incidence of type 2 diabetes than lean individuals, but they also developed more infections than non-obese individuals," she said.

Blood samples taken from these patients revealed that both NKT cells and iNKT cells were decreased, and subsequent studies of fat tissue from a group of obese patients who had lost weight following bariatric surgery showed that iNKT cells had increased to normal levels.

In this new research, conducted with colleagues at BIDMC, St. Vincent's University Hospital, and Trinity College Dublin, the authors conducted a series of animal experiments to test their hypothesis that iNKT cells play a role in fat tissue regulation and protect against the development of inflammation and the metabolic syndrome.

Previous research by Professors Mark Exley and Cliona O'Farrelly had described large numbers of iNKT cells in human and mouse liver tissue; the group therefore needed to ascertain that, like humans, mice also harboured these cells in fat .

"We found loads of them," Dr Lynch said.

The research team then put the mice on a high-fat diet and studied the outcome.

"Similar to the human subjects we had previously studied, the animals lost their iNKT cells when they became obese," Lynch said. "Once we took them off this diet and put them back on a normal standard-fat diet, they lost the weight -- and their iNKT cells increased."

In the next experiment, the authors set out to better understand the exact role of the iNKT cells by examining two strains of mice, both of which are deficient in iNKT cells, and a group of control mice, all on a high-fat diet.

Although all the animals grew obese, the iNKT-deficient mice grew 30 percent fatter than the control animals and developed the mouse equivalent of type 2 diabetes over just six weeks. The mice also had greatly increased triglyceride levels, larger fat cells, and fatty liver disease.

Next, the authors removed iNKT cells from a normal mouse and injected them into obese NKT-deficient mice.

"We actually reversed the diabetes, and even though the mice continued to eat a high-fat diet, they lost one to two grams of weight [normal mouse weight being 20 to 25 grams] and exhibited a host of features that suggested reduced inflammation, including improved insulin sensitivity, lower triglycerides and leptin, and shrunken adipocytes," Dr Lynch said.

Finally, in order to demonstrate if the remaining diminished pool of iNKT cells in obesity could be activated to improve metabolism, the scientists tested aGC, a lipid known to activate iNKT cells. They found that administering a single dose of aGC caused a dramatic improvement in metabolism and fatty liver disease, loss of much of the weight gained, and reversal of diabetes in the obese animals.

"aGC has been tested in clinical trials for the treatment of certain cancers, including melanoma, and proven safe and produced few side effects in humans," said Exley. "The effect of NKT stimulation, whether by aGC or other means, on weight loss, obesity, and metabolic disorder has not been investigated until now and may provide a new avenue for the treatment of obesity and metabolic syndrome, which have now reached epidemic proportions worldwide."

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Story Source:

The above story is reprinted from materials provided by Trinity College Dublin, via AlphaGalileo.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Lydia Lynch, Michael Nowak, Bindu Varghese, Justice Clark, Andrew?E. Hogan, Vasillis Toxavidis, Steven?P. Balk, Donal O?Shea, Cliona O?Farrelly, Mark?A. Exley. Adipose Tissue Invariant NKT Cells Protect against Diet-Induced Obesity and Metabolic Disorder through Regulatory Cytokine Production. Immunity, 2012; 37 (3): 574 DOI: 10.1016/j.immuni.2012.06.016

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/most_popular/~3/9Ka4Pz8mA9A/121001095522.htm

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Nissan buys back Leafs under Arizona Lemon Law

Some Nissan Leaf drivers are returning their vehicles for a full refund, citing reduced range and battery power. But Nissan insists that such problems are rare, and that Leaf owners are among the country's most satisfied car owners.?

By Nikki Gordon-Bloomfield,?Guest blogger / September 30, 2012

In this March 2012 file photo, a Nissan Leaf tops off it's battery in Central Point, Ore., at one of the charging stations along Interstate 5. Some Arizona Leaf owners are returning their vehicles in exchange for a full refund under that state's Lemon Law. Nissan insists such returns are isolated occurrences, and not an emerging trend of dissatisfaction among Leaf owners.

Jeff Barnard/AP/FIle

Enlarge

Yesterday, a local news station in Phoenix, Arizona, confirmed that?Nissan?has agreed to buy back wilting Leaf?electric?cars?from their owners under the state?s Lemon Law.

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The news comes less than a week after Mark Perry, Nissan North America?s product planning and?advanced?technology?director, blamed the premature range loss suffered by some Leaf owners in hot states on?excessive high mileage.?

As CBS5 in Phoenix reports, Andrea and Mason Convey, and Scott Yarosh, are among those who are selling their Leafs back to Nissan.

Earlier this year, Convey and Yarosh?s cars were among seven Leafs taken to Nissan?s testing facility in Case Grande for?official Nissan examination, after continued complaints to the automaker.?

?I had to get rid of it,? Yarosh told CBS5. ?When I turned my?car?in, I was only able to get 42 miles on a single charge,? he explained, pointing out that his work commute was 45 miles.?

After just 15 months of ownership, three of the capacity bars on his Leaf?s dashboard had disappeared, representing a 27.5 percent loss in battery capacity.

Unable to drive his car to work, Yarosh turned his car in, and was hit by almost $700 in fees. A week later, Nissan gave him a full refund.?

?I think they?re trying to get me to shut up to be honest, to keep my mouth shut,? he said.

The Conveys are similarly suspicious.

?We?re not sure if this is the beginning of their admission that there is an issue or if this is them trying to quiet a few isolated individuals,? Mason Convey said. ?It?s been a long four months and there?s still a lot of bitterness still left over from the way they treated us.?

Other Leaf owners have also accepted a buy-back offer from Nissan, including Randy Miller, owner of the appropriately named WiltingLeaf.com blog.

?One thing I will point out is that the repurchase price is based on the mileage (reasonable use charge according to AZ Lemon Law), and that mileage is based on the first complaint to Nissan,? Miller wrote. ?So if you are a Leaf owner and are having range / battery capacity issues, be sure to file a complaint with the BBB Auto Line as soon as possible.?

According to Nissan spokeswoman Katherine Zachary, the decision to buy back cars isn't an admittance of fault on?Nissan's?part: it's a way to keep customers happy.

"Nissan works hard to satisfy individual customers when they express unhappiness with their ownership experience," she told us. "In the interest of customer satisfaction, Nissan has recently repurchased two customer vehicles as a good will gesture. The company's investigation has determined that there is no defect with the Nissan Leaf, but we did use a buyback formula modelled on an Arizona state repurchase law, given its established criteria."

Reiterating that Nissan believes the issue of premature battery loss to be confined to a small number of cases, and that globally, Nissan Leaf customers remain one of the most satisfied car owners in the world, Zachary added, "Remember, this is a small group of owners we're talking about, and it's not a pervasive problem. It's a combination of high temperatures and high miles in a small number of cases."

Nissan has not made an official statement on the subject of the first??class-action lawsuit now filed against Nissan in California?for the same complaint.?

The Christian Science Monitor has assembled a diverse group of the best auto bloggers out there. Our guest bloggers are not employed or directed by the Monitor and the views expressed are the bloggers' own, as is responsibility for the content of their blogs. To contact us about a blogger,?click here.?To add or view a comment on a guest blog, please go to the blogger's own site by clicking on the link in the blog description box above.

Source: http://rss.csmonitor.com/~r/feeds/csm/~3/4fSGOB330Ik/Nissan-buys-back-Leafs-under-Arizona-Lemon-Law

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Well: Not a Cancer Survivor

I am not a cancer survivor, and neither are the women in my cancer support group.

Mary feels that cancer was a ?blip? in her past that no longer defines her. Diane is a survivor, but not of cancer; she is ?a survivor of treatments of cancer.?

Patricia and Judy are not survivors, because they are undergoing their first treatments and have no idea how effective they will be. Not a survivor either, Sarah braces herself for the time ? not if, but when ? the cancer will return. And there is Allison, who, like me, feels put off by the word ?survivor?; somehow the term sounds too heroic to claim for ourselves.

In newspaper articles, on TV shows and Web sites, and at social gatherings, many people with cancer define themselves as cancer survivors. The term is meant to be optimistic, suggesting that such people have beaten cancer, defeated the disease. Through a valiant struggle to endure, they have managed to get through the trauma of cancer and emerge on the other side, perhaps sadder but wiser and possibly even better equipped for existence, for they are now attuned to the precious, precarious nature of human life.

While I can only congratulate such people, surely there are others (besides the members of my support group) who cringe at adopting such an identity ? and for a number of reasons. Does the celebration of the triumphant cancer survivor cast those who died from the disease in the role of victims who somehow failed to attain the requisite resiliency to overcome it? An American propensity to circulate stories of valiant individuals triumphing over great odds must make people coping with recurrent, chronic or terminal illness feel like duds. And even for those patients with cancers that can be cured, claiming to be a survivor might feel dangerous ? like a jinx, a sign of the sort of chutzpah or hubris that could bring about dire reprisals from the powers that be.

Despite all the hype surrounding the ?war against cancer,? many cancers remain incurable, and the people coping with them need some other terms to describe their sense of themselves. Approximately 40 percent of the American population will get a form of cancer; half of them (roughly 20 percent) do not survive. There must be (and must have been) quite a few people who have known themselves not to be survivors. What should we call patients up against these numbers? If some of us are not cancer survivors before our dying, are we cancer contenders? Cancer lifers, cancer dealers, cancer mavens, grits? As I eagerly await any and all suggestions, I ponder the various lexicons that mystify or vex people trying to keep a sense of self intact after dire diagnoses and sometimes draconian treatments.

If commonly used words pose a problem, so do arcane, ugly and incomprehensible lexicons that may serve the needs of medical specialists, but prove trying for many patients who have no idea what ?creatinine? or ?platelets,? ?neutrophil counts? or ?ecog status? really mean. In the 2010 novel ?The Sickness,? by the Venezuelan writer Alberto Barrera Tyszka, a physician whose father is dying of lung cancer ?finds the clinical terms unbearable,? forming ?part of a pretentious, useless dictionary?:

neoplasty, exeresis staphylococcal empyema
pleural empyema anastomosis iliocolostomy
biopsy haemostasis prothesis laparotomy
ischemia lithiasis.

Just as incomprehensible, the acronyms resounding in hospital halls and rooms ? PIC, CT, BRAC, PSA, GOG, CA-125, NPO, PEG, NG, PTN, PK, HER2, DCIS, LCIS ? need to be translated for the ordinary listener, or they degenerate into an unpalatable alphabet soup.

And then consider the sometimes hilariously inappropriate languages of friends and relatives responding to a diagnosis, which happily make the members of my support group laugh.

?What a tragedy for your children,? Diane reports one acquaintance exclaiming ? an excellent example of what I would call predatory pre-grieving.

Or ?You should have gone to M.D. Anderson,? which is a nice instance of the know-it-all with information after the fact.

Or ?You look great. Just eat your flaxseeds, and you?ll be fine,? a rejoinder of indefatigable optimism that some of us find off-putting.

Even frequently used rubrics can mask complex realities. Take, for example, our usual categories of ?remission? versus ?recurrence.? Often patients who feel perfectly well believe they are in a remission until their physician informs them that some sort of blood test or body scan proves that the cancer has returned.

The language of remission and recurrence makes it seem as if the cancer is gone, then back. But the hide-and-seek in many case histories suggests that while a number of cancers recede, they do not disappear. Some people live during long periods of contestation in which treatments continue to exert their sway, but the cancer does too.

Perhaps we need a word for that murky in-between zone that a number of us inhabit daily.

Susan Gubar is a distinguished emerita professor of English at Indiana University and the author of ?Memoir of a Debulked Woman,? which explores her experience with ovarian cancer and the need for better detection tools and treatments.

Suleika Jaouad, whose ?Life, Interrupted? column usually appears on Thursdays, will return in two weeks.

Source: http://well.blogs.nytimes.com/2012/09/06/not-a-cancer-survivor/?partner=rss&emc=rss

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Ginger Pear-Apple Crunch Recipe

One of my neighbors has some pear and apple trees, and invited me over to pick some in exchange for a pie or a crisp. When I got to his yard, I was amazed at the bounty of fruits on several trees. I?ll be picking for a few weeks as each variety ripens. I can hardly wait!

I came up with a list of different ways to serve the apples and pears, and the idea for a feast using these fruits in every course. Here is the first recipe I decided to try out. It was so good, I had to make a second one, so that I?d have enough for me after giving some to my neighbor.

Note: While you can use any variety of pears and apples for this recipe, I used Pink Pearl apples, Seckel pears, and Bosc pears. The Seckels, which my neighbor dubbed Sucre, are the sweetest pear I?ve ever eaten, very juicy and sugary. WOW. When you?re picking, ripe fruits should come off the stem easily, with almost no pressure. If you have to twist the fruit off the stem, it?s not ripe yet.

Ginger Pear-Apple Crunch

For the filling:

4 Pink Pearl or other apples
6 Seckel pears (about 6 cups total sliced fruit)
2 Bosc pears
? cup dried cranberries
? cup Golden or other raisins
? cup Agave syrup
1 tsp. vanilla extract
1-2 tsp. grated fresh ginger
? tsp. Kashmiri Masala ( see recipe below)
1 tsp. lavender buds
8 cardamom pods, split, seeds removed
1 Tbls. flour (whole wheat pastry is best)
juice of 1 lemon (grate the rind for the topping before cutting the lemon)

1) Peel and core the apples and pears

2) In a large bowl, carefully toss all the ingredients.

3) Spread the filling evenly in a buttered 8? x 11? baking dish.

4) Preheat oven to 375? F while making the topping.

For the crunch topping:

1 cup whole-wheat pastry flour
? cup rolled oats
? cup packed light brown or white sugar
? cup (1 stick) cold unsalted butter
grated rind of one lemon

1) Mix the flour, oats, and sugar, and lemon rind in a medium bowl. Cut the butter into small pieces and work in with a pastry blender to make uniform crumbs. Note: When I first made this, I misread my notes and used only half a stick of butter?and it came out fine.

2) Spread the topping evenly over the fruits. Bake for 45 minutes, or until fruit is soft and topping is bubbly and golden.

3) Cool on a rack for 10 minutes before serving. Now, go sit in corner by yourself--making sure there are no partners, small children or pets to disturb you--and eat this with vanilla ice cream or plain cream or coconut milk. Hide the leftovers for another time.

Kashmiri Masala

8 green cardamom pods
1 Tbsp. black peppercorns
2 tsp. whole cloves
1 cinnamon stick
2 Tbsp. cumin seeds
1 tsp. freshly grated nutmeg

1) Remove the seeds from the cardamom pods and crush them with a mortar and pestle or the end of a rolling pin. Break the cinnamon into several small pieces.
2) Dry-roast all the spices except the nutmeg in a small skillet for 3 to 4 minutes. Let cool.

3) Grind the spices, including nutmeg, to a fine powder with a mortar and pestle or an electric grinder (such as a clean coffee grinder). If necessary, put the spices through a strainer to remove larger pieces. Place them in a jar and store in a cool, dark place.

Source: http://www.bellaonline.com/articles/art40584.asp

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